Medulloblastoma — SickKids 2016
Overview
Whole-genome sequencing of 33 patient-matched primary/recurrence medulloblastoma (MBL) pairs (46 total human samples) from a multi-institutional pediatric brain tumor cohort, combined with whole-exome sequencing of 3 FFPE pairs. The study was designed to characterize clonal evolution between diagnosis and relapse. Raw sequencing deposited at EGA (EGAD00001000946). Reference genome GRCh37/hg19.
Composition
- 15 matched primary + recurrent pairs with germline controls: whole-genome-seq.
- 18 matched primary + recurrent pairs without germline: whole-genome-seq.
- 10 recurrence-only samples with germline: whole-genome-seq.
- 3 FFPE matched pairs: whole-exome-seq (Nextera Rapid Capture).
- Total human samples: 46 from 33 patients.
- All four medulloblastoma molecular subgroups represented (Shh, Wnt, Group 3, Group 4).
- Cancer type: MBL (medulloblastoma).
Assays / panels (linked)
- whole-genome-seq (BWA / GRCh37-lite alignment; Strelka + MutationSeq for SNVs)
- whole-exome-seq (Nextera Rapid Capture for FFPE pairs)
- targeted-deep-amplicon-seq (192 patient-specific SNVs across 20 patients)
- strelka / varscan (variant calling)
- titan-cna (allele-specific copy-number)
- pyclone / EXPANDS (clonal-prevalence inference)
Papers using this cohort
- PMID:26760213 — Morrissy et al. 2016, “Divergent clonal selection dominates medulloblastoma at recurrence,” Nature.
Notable findings derived from this cohort
- On average only 11.8% of somatic SNVs/indels are shared between matched diagnostic and recurrent tumors, demonstrating massive clonal divergence at relapse PMID:26760213.
- Somatic mutational burden increases ~5-fold post-therapy in 13/15 patient pairs (P = 2.7 × 10⁻⁴); structural-variant burden also rises PMID:26760213.
- Recurrence arises from pre-existing minor subclones (<5% at diagnosis) confirmed in 16/20 patients by ultra-deep targeted resequencing PMID:26760213.
- TP53-pathway alterations (including TP53 mutations) and DYNC1H1/chr14q deficits together affect 79% of recurrent Shh medulloblastoma PMID:26760213.
- Drug-actionable targets at recurrence differ from those at diagnosis in the majority of patients (5/9 with any actionable alteration); 2/9 had a complete actionable-target switch PMID:26760213.
- Branched clonal evolution demonstrated in all 14 germline-matched cases; Group 4 tumors significantly more heterogeneous at recurrence (Shannon Index, P = 0.029) PMID:26760213.
Sources
- PMID:26760213
- EGA: EGAD00001000946
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