Johns Hopkins Pancreatic Neuroendocrine Tumor Exome Sequencing
Overview
Whole-exome sequencing study of 68 sporadic, non-familial pancreatic neuroendocrine tumors (PanNETs) from Johns Hopkins University and Memorial Sloan Kettering Cancer Center. The study used a two-stage design: 10 tumors for whole-exome discovery (Illumina GAIIx with SureSelect capture, mean 101x coverage) and 58 additional tumors for targeted Sanger validation. Familial syndrome-associated tumors and small cell/large cell neuroendocrine carcinomas were excluded. All tumors were microdissected to >80% neoplastic cellularity.
Composition
- Cancer type: PANET
- 68 sporadic PanNETs: 10 discovery (WES) + 58 validation (Sanger)
- Key clinical fields: survival, stage, grade, Ki-67 index, metastatic status
- Assays: whole-exome-seq (discovery set) and sanger-sequencing (validation set)
Assays / panels (linked)
- whole-exome-seq — Illumina GAIIx, SureSelect capture, ~18,000 genes
- sanger-sequencing — targeted validation in 58 additional cases
Papers using this cohort
- PMID:21252315 — Jiao et al. 2011, exome sequencing of PanNETs identifying MEN1/DAXX/ATRX driver landscape
Notable findings derived from this cohort
- MEN1 mutated in 44.1% (30/68) of PanNETs; 25/30 mutations were inactivating (indels, nonsense, splice-site) PMID:21252315
- DAXX mutated in 25% and ATRX in 17.6%; mutations were mutually exclusive; combined DAXX/ATRX pathway alteration rate 42.6% PMID:21252315
- mTOR pathway genes mutated in 14%: PTEN 7.3%, TSC2 8.8%, PIK3CA 1.4% PMID:21252315
- MEN1 and DAXX/ATRX mutations associated with significantly better prognosis: 100% survival at 10 years vs. >60% mortality within 5 years in mutation-negative patients PMID:21252315
- PanNETs have a distinct genetic landscape from pancreatic ductal adenocarcinoma: KRAS 0% and TP53 3% in PanNETs vs. ~100% and ~85% in PDAC PMID:21252315
- Mean 16 somatic mutations per tumor; 91% of discovery-set mutations validated by Sanger sequencing PMID:21252315
Sources
- PMID:21252315 — Jiao et al., “DAXX/ATRX, MEN1, and mTOR pathway genes are frequently altered in pancreatic neuroendocrine tumors,” Science 2011
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