PGR
Overview
PGR encodes the progesterone receptor, a nuclear hormone receptor that mediates progesterone signaling and plays a central role in uterine biology. In endometrial cancer, PGR expression is a marker of hormonal responsiveness and is characteristically elevated in the lower-grade, copy-number-low endometrioid subtype. Loss of PGR expression is associated with more aggressive, hormone-insensitive disease.
Alterations observed in the corpus
- Protein expression increased in the copy-number-low endometrial cancer cluster by RPPA, consistent with hormonal-therapy responsiveness of this endometrioid subgroup PMID:23636398
- PR expression correlates with ER expression across CRISPR rat mammary tumor models; PGR transcript appears in the 58-gene set responsive to fulvestrant in rat tumors and in human GSE5462/GSE71791 datasets PMID:26437033
- PGR (progesterone receptor) discussed as a candidate alternative activator of AR-pathway output in mCRPC tumors with AR activity but absent AR protein expression, alongside NR3C1 (GR) and ESR1 (ER-alpha). PMID:26928463
Cancer types (linked)
- UCEC (Uterine Corpus Endometrial Carcinoma): PGR protein upregulation is a feature of the copy-number-low MSS endometrioid molecular subgroup, which is associated with better prognosis and potential sensitivity to progestin-based hormonal therapy PMID:23636398
Co-occurrence and mutual exclusivity
- Elevated PGR expression co-occurs with the copy-number-low endometrioid molecular subtype, which is defined by CTNNB1 mutations and low overall genomic instability PMID:23636398
Therapeutic relevance
- High PGR expression in copy-number-low endometrioid tumors supports the use of progestin-based hormonal therapies in this subgroup, as proposed by TCGA endometrial cancer molecular classification PMID:23636398
Open questions
- Whether PGR protein levels (RPPA) correlate with clinical response to progestin therapy in prospective endometrial cancer cohorts remains to be demonstrated.
Sources
This page was processed by crosslinker on 2026-05-14. - PMID:26437033
This page was processed by crosslinker on 2026-05-14. - PMID:26928463
This page was processed by wiki-cli on 2026-05-14.