RICTOR
Overview
RICTOR is a core component of the mTORC2 complex. In the corpus it is highlighted as a subclonal resistance driver detectable by ctDNA in advanced NSCLC.
Alterations observed in the corpus
- Enriched among ctDNA-only alterations (not detected by time-matched tissue sequencing) in the 1,127-patient prospective NSCLC ctDNA cohort; flagged as a subclonal driver of resistance associated with short survival PMID:36357680.
- Recurrent copy number amplifications and mutations in RICTOR detected in melanoma WES cohort (147 tumors, Yale); identified as significantly mutated gene PMID:22842228
- RICTOR high expression supported mTOR-inhibitor (everolimus) recommendations in neuroendocrine neoplasms PN4 and PN25 within a WGTA-guided precision oncology cohort PMID:24326773
- RICTOR (MTORC2 component) part of PI3K/AKT/MTOR pathway alterations present in 84% of ULMS+STLMS C1 vs 44% of STLMS C2 sarcoma; nominates dual PI3K/MTOR or TORC1/TORC2 inhibitors as therapeutic strategy PMID:29100075
Cancer types (linked)
- Non-small-cell lung cancer (LUAD, LUSC) — ctDNA-only subclonal resistance driver PMID:36357680.
Co-occurrence and mutual exclusivity
- Appears alongside PIK3CA in the ctDNA-only subclonal resistance spectrum PMID:36357680.
Therapeutic relevance
- No direct therapeutic link reported; flagged as potentially relevant to resistance management PMID:36357680.
Open questions
- Whether ctDNA-only RICTOR subclonal alterations should directly alter NSCLC treatment selection remains an open cross-paper question PMID:36357680.
Sources
This page was processed by crosslinker on 2026-05-09. - PMID:22842228
This page was processed by crosslinker on 2026-05-09. - PMID:24326773
This page was processed by crosslinker on 2026-05-09. - PMID:29100075
This page was processed by wiki-cli on 2026-05-15.