CACNA1D
Overview
CACNA1D encodes the alpha-1D subunit of a voltage-dependent L-type calcium channel (Cav1.3). In cancer genomics, CACNA1D has been observed as a somatically mutated gene in gastric cancer, with the p.V529G missense variant notable for being shared between two patients in a multi-region sequencing study, suggesting either a convergent selective pressure or a shared mutational mechanism.
Alterations observed in the corpus
- Missense p.V529G variant shared between two distinct gastric cancer patients (Pt1 and Pt2) in a multi-region WGS clonal evolution study of 294-patient Tianjin WES cohort PMID:25583476
Cancer types (linked)
- Gastric cancer (STAD): The p.V529G variant observed in two independent patients in a clonal architecture study; its recurrence across patients suggests potential selective advantage PMID:25583476
Co-occurrence and mutual exclusivity
- In Pt1: co-occurs with KRAS p.G13D, PIK3CA p.E542K, and BRCA2 mutations across separate tumor clones PMID:25583476
- In Pt2: co-occurs with SETBP1 p.S944N, AKAP9 mutation, and CDK6 amplification PMID:25583476
Therapeutic relevance
- No direct therapeutic targeting of CACNA1D reported in the corpus; the shared mutation across patients may merit investigation as a candidate driver.
Open questions
- Whether p.V529G is functionally activating or loss-of-function in the gastric cancer context is unknown.
- The mechanism by which independent gastric cancer patients share this exact variant is not explained in the source paper.
- Broader prevalence in population-scale gastric cancer datasets is not reported in the corpus.
Sources
This page was processed by crosslinker on 2026-05-14.