GPS2

Overview

GPS2 (G Protein Pathway Suppressor 2) encodes a component of the N-CoR (nuclear receptor co-repressor) transcriptional repressor complex. In medulloblastoma, GPS2 harbors somatic missense mutations within the NCOR2 interaction domain (amino acids 53–90) specifically in Group 3 tumors, implicating disruption of the N-CoR co-repressor complex in this aggressive medulloblastoma subgroup.

Alterations observed in the corpus

  • Missense mutations within the NCOR2-interaction domain (aa 53–90) identified in Group 3 medulloblastoma by WES (Broad, 92 tumors) PMID:22820256
  • Identified in a 173-gene targeted sequencing panel breast cancer study (n=2,433) as part of the mutational landscape analysis PMID:27161491

Cancer types (linked)

  • MB (medulloblastoma): Mutations in Group 3 subtype; disrupts N-CoR/NCOR2 co-repressor complex; subtype-specific MutSig candidate PMID:22820256

Co-occurrence and mutual exclusivity

  • Mutations cluster in the NCOR2 interaction domain, suggesting disruption of the same GPS2–NCOR2 protein interface as LDB1 and other N-CoR complex members altered in medulloblastoma PMID:22820256

Therapeutic relevance

  • N-CoR complex disruption across multiple components (GPS2, LDB1, NCOR2) in medulloblastoma may indicate sensitivity to epigenetic therapies targeting nuclear co-repressor pathways.

Open questions

  • Whether GPS2 mutations are sufficient to drive Group 3 medulloblastoma or require co-occurring alterations remains unknown.
  • Functional validation of GPS2 missense mutations on N-CoR complex activity not yet performed.

Sources

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