USH2A
Overview
USH2A (Usherin) encodes a large extracellular matrix protein involved in cell adhesion; it is best known as a cause of Usher syndrome but has been observed as a recurrently mutated gene in anaplastic thyroid carcinoma.
Alterations observed in the corpus
- USH2A was identified as a top-5 recurrently mutated gene by SeqSig FDR analysis in anaplastic thyroid carcinoma (ATC); previously described in ATC in independent studies PMID:38412093.
- USH2A is significantly over-expressed in the undifferentiated nC3 cluster enriched in high-risk neuroblastoma (FDR <0.01, Welch’s t-test), along with progenitor markers BCL11A, NTRK2, and SOX6 in a single-nuclei RNA-seq study (11 tumors, Smart-Seq2). PMID:34493726
- Mutations in 9.2% (6/65) of triple-negative breast cancers in WGS cohort; gene involved in actin cytoskeletal functions PMID:22495314
Cancer types (linked)
- THPA — top-5 SeqSig recurrent mutation (FDR-controlled) in ATC PMID:38412093.
- NBL — over-expressed in undifferentiated nC3 high-risk neuroblastoma cluster, as part of the progenitor gene program shared with the postnatal adrenal hC1 progenitor. PMID:34493726
Co-occurrence and mutual exclusivity
- USH2A mutations co-occur with other ATC drivers (TP53, BRAF V600E, CDKN2A deletions) in the ATC genomic landscape PMID:38412093.
- Co-expressed with MYCN, ALK, SOX6, TP63, LGR5 in undifferentiated nC3 neuroblastoma cluster. PMID:34493726
Therapeutic relevance
- No direct targeted therapy reported in the corpus.
Open questions
- The functional role of USH2A mutations in ATC pathogenesis (driver vs. passenger) is not resolved in the corpus PMID:38412093.
Sources
This page was processed by entity-page-writer on 2026-04-15. - PMID:22495314
This page was processed by wiki-cli on 2026-05-06.